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Characterization of the lipopolysaccharide induced model of Parkinson's  disease: Role of oxidative stress and neuroinflammation - ScienceDirect
Characterization of the lipopolysaccharide induced model of Parkinson's disease: Role of oxidative stress and neuroinflammation - ScienceDirect

The phosphodiesterase 10 inhibitor papaverine exerts anti-inflammatory and  neuroprotective effects via the PKA signaling pathway in neuroinflammation  and Parkinson's disease mouse models | Journal of Neuroinflammation | Full  Text
The phosphodiesterase 10 inhibitor papaverine exerts anti-inflammatory and neuroprotective effects via the PKA signaling pathway in neuroinflammation and Parkinson's disease mouse models | Journal of Neuroinflammation | Full Text

Neuronal Mitochondria Modulation of LPS-Induced Neuroinflammation | Journal  of Neuroscience
Neuronal Mitochondria Modulation of LPS-Induced Neuroinflammation | Journal of Neuroscience

Salidroside ameliorates Parkinson's disease by inhibiting NLRP3-dependent  pyroptosis - Figure f10 | Aging
Salidroside ameliorates Parkinson's disease by inhibiting NLRP3-dependent pyroptosis - Figure f10 | Aging

PDF] Inflammatory Animal Model for Parkinson's Disease: The Intranigral  Injection of LPS Induced the Inflammatory Process along with the Selective  Degeneration of Nigrostriatal Dopaminergic Neurons | Semantic Scholar
PDF] Inflammatory Animal Model for Parkinson's Disease: The Intranigral Injection of LPS Induced the Inflammatory Process along with the Selective Degeneration of Nigrostriatal Dopaminergic Neurons | Semantic Scholar

The Endotoxin-Induced Neuroinflammation Model of Parkinson's Disease
The Endotoxin-Induced Neuroinflammation Model of Parkinson's Disease

Frontiers | The Gut–Brain Axis and Its Relation to Parkinson's Disease: A  Review
Frontiers | The Gut–Brain Axis and Its Relation to Parkinson's Disease: A Review

Experimental Neurobiology
Experimental Neurobiology

Lipopolysaccharide animal models of Parkinson's disease: Recent progress  and relevance to clinical disease - ScienceDirect
Lipopolysaccharide animal models of Parkinson's disease: Recent progress and relevance to clinical disease - ScienceDirect

MicroRNA‐3473b regulates the expression of TREM2/ULK1 and inhibits  autophagy in inflammatory pathogenesis of Parkinson disease - Lv - 2021 -  Journal of Neurochemistry - Wiley Online Library
MicroRNA‐3473b regulates the expression of TREM2/ULK1 and inhibits autophagy in inflammatory pathogenesis of Parkinson disease - Lv - 2021 - Journal of Neurochemistry - Wiley Online Library

LPS-Induced Neuroinflammation Model - Creative Biolabs
LPS-Induced Neuroinflammation Model - Creative Biolabs

Lipopolysaccharide Animal Models for Parkinson's Disease
Lipopolysaccharide Animal Models for Parkinson's Disease

Chronic neuroinflammation generated by a single LPS injection (15 × 10⁶...  | Download Scientific Diagram
Chronic neuroinflammation generated by a single LPS injection (15 × 10⁶... | Download Scientific Diagram

Lipopolysaccharide animal models of Parkinson's disease: Recent progress  and relevance to clinical disease - ScienceDirect
Lipopolysaccharide animal models of Parkinson's disease: Recent progress and relevance to clinical disease - ScienceDirect

Is Porphyromonas gingivalis involved in Parkinson's disease? | SpringerLink
Is Porphyromonas gingivalis involved in Parkinson's disease? | SpringerLink

Parkinson's Disease-Related Proteins PINK1 and Parkin Repress Mitochondrial  Antigen Presentation: Cell
Parkinson's Disease-Related Proteins PINK1 and Parkin Repress Mitochondrial Antigen Presentation: Cell

Intranasal LPS-Mediated Parkinson's Model Challenges the Pathogenesis of  Nasal Cavity and Environmental Toxins | PLOS ONE
Intranasal LPS-Mediated Parkinson's Model Challenges the Pathogenesis of Nasal Cavity and Environmental Toxins | PLOS ONE

Is Porphyromonas gingivalis involved in Parkinson's disease? | SpringerLink
Is Porphyromonas gingivalis involved in Parkinson's disease? | SpringerLink

DL‑3‑n‑butylphthalide reduces microglial activation in lipopolysaccharide‑induced  Parkinson's disease model mice
DL‑3‑n‑butylphthalide reduces microglial activation in lipopolysaccharide‑induced Parkinson's disease model mice

IJMS | Free Full-Text | Licochalcone A Prevents the Loss of Dopaminergic  Neurons by Inhibiting Microglial Activation in Lipopolysaccharide (LPS)-Induced  Parkinson's Disease Models
IJMS | Free Full-Text | Licochalcone A Prevents the Loss of Dopaminergic Neurons by Inhibiting Microglial Activation in Lipopolysaccharide (LPS)-Induced Parkinson's Disease Models

LPS induces chronic neuroinflammation with heterogeneous densities of... |  Download Scientific Diagram
LPS induces chronic neuroinflammation with heterogeneous densities of... | Download Scientific Diagram

Urate inhibits microglia activation to protect neurons in an LPS-induced  model of Parkinson's disease | Journal of Neuroinflammation | Full Text
Urate inhibits microglia activation to protect neurons in an LPS-induced model of Parkinson's disease | Journal of Neuroinflammation | Full Text

Impact of Aging and Gut Microbiota on Neuroinflammation and Parkinson's  Disease | PerkinElmer
Impact of Aging and Gut Microbiota on Neuroinflammation and Parkinson's Disease | PerkinElmer

Lipopolysaccharide animal models of Parkinson's disease: Recent progress  and relevance to clinical disease - ScienceDirect
Lipopolysaccharide animal models of Parkinson's disease: Recent progress and relevance to clinical disease - ScienceDirect

Synergistic Dopaminergic Neurotoxicity of the Pesticide Rotenone and  Inflammogen Lipopolysaccharide: Relevance to the Etiology of Parkinson's  Disease | Journal of Neuroscience
Synergistic Dopaminergic Neurotoxicity of the Pesticide Rotenone and Inflammogen Lipopolysaccharide: Relevance to the Etiology of Parkinson's Disease | Journal of Neuroscience

Intranasal LPS-Mediated Parkinson's Model Challenges the Pathogenesis of  Nasal Cavity and Environmental Toxins | PLOS ONE
Intranasal LPS-Mediated Parkinson's Model Challenges the Pathogenesis of Nasal Cavity and Environmental Toxins | PLOS ONE